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Fish oil potentiates excitotoxicity?

Hi all,
Can any of you clarify whether supplementation with fish oil is beneficial for excess glutamate (e.g. benzo withdrawal)? Fish oil seems to be widely suggested as a harmless, neuroprotective substance, specifically so during benzo withdrawal, but the following papers seem to conclude otherwise;
  1. Increasing glutamate activity
"Glutamate DHA enhances glutamatergic synaptic activities with concomitant increases in synapsin and glutamate receptor subunit expression in hippocampal neurons (Kim et al., 2011b). Spontaneous synaptic activity is signifi cantly increased due to enhanced glutamatergic activity in DHA-supplemented neurons. On the other hand, lack of DHA results in inhibition of synaptogenesis, decreases in synapsins and glutamate receptor subunits, and impairment of long-term potentiation in hippocampal neurons (Cao et al., 2009). DHA modulates the activities of glutamate transporters (GLT1, GLAST, and EAAC1). DHA stimulates GLT1 and EAAC1 through a mechanism that requires extracellular Ca2+, CaM kinase II, and protein kinase C but not protein kinase A. "
"DHA inhibits GABA receptor-mediated responses in cultured neural cells in a concentration and time-dependent manner. It is shown that the γ subunit is essential for the potentiation of GABA induced Cl− channel activity and effect on desensitization kinetics of the GABAA receptor (Nabekura et al., 1998). DHA accelerates desensitization after the peak of the GABA-induced current, potentiates the peak amplitude of GABA response, and gradually suppresses the peak amplitude of GABA response (Nabekura et al., 1998). It is suggested that the effect of DHA on GABA receptor is due to the effect on the lipid microenvironment for the GABA receptors (Nabekura et al., 1998)."
(https://pdfs.semanticscholar.org/9002/0e650bb5a23982b6c2550c990c150293e0c9.pdf)
  1. Decreasing GABA activity
"DHA, as well as AA, may play an important role in modulating neuronal excitability by reducing the IGABA and Igly, and potentiating N-methyl-D-aspartate receptor-mediated responses in the SN."
https://www.ncbi.nlm.nih.gov/pubmed/8867135
  1. Another paper on how DHA inhibits GABA-receptor mediated responses and enhances glutamatergic synaptic activities (see "EFFECTS OF DHA ON NEUROTRANSMITTERS"); https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3792211/
Could anyone elaborate on this?
Thank you all.
submitted by RelevantBarnacle to Nootropics

Fish oil potentiates excitotoxicity and accelerates gaba-A desensitization?

Hi all,
Can any of you clarify whether supplementation with fish oil is beneficial for excess glutamate (e.g. benzo withdrawal)? Fish oil seems to be widely suggested as a harmless, neuroprotective substance, specifically so during benzo withdrawal, but the following papers seem to conclude otherwise;
  1. Increasing glutamate activity
"Glutamate DHA enhances glutamatergic synaptic activities with concomitant increases in synapsin and glutamate receptor subunit expression in hippocampal neurons (Kim et al., 2011b). Spontaneous synaptic activity is signifi cantly increased due to enhanced glutamatergic activity in DHA-supplemented neurons. On the other hand, lack of DHA results in inhibition of synaptogenesis, decreases in synapsins and glutamate receptor subunits, and impairment of long-term potentiation in hippocampal neurons (Cao et al., 2009). DHA modulates the activities of glutamate transporters (GLT1, GLAST, and EAAC1). DHA stimulates GLT1 and EAAC1 through a mechanism that requires extracellular Ca2+, CaM kinase II, and protein kinase C but not protein kinase A. "
"DHA inhibits GABA receptor-mediated responses in cultured neural cells in a concentration and time-dependent manner. It is shown that the γ subunit is essential for the potentiation of GABA induced Cl− channel activity and effect on desensitization kinetics of the GABAA receptor (Nabekura et al., 1998). DHA accelerates desensitization after the peak of the GABA-induced current, potentiates the peak amplitude of GABA response, and gradually suppresses the peak amplitude of GABA response (Nabekura et al., 1998). It is suggested that the effect of DHA on GABA receptor is due to the effect on the lipid microenvironment for the GABA receptors (Nabekura et al., 1998)."
(https://pdfs.semanticscholar.org/9002/0e650bb5a23982b6c2550c990c150293e0c9.pdf)
  1. Decreasing GABA activity
"DHA, as well as AA, may play an important role in modulating neuronal excitability by reducing the IGABA and Igly, and potentiating N-methyl-D-aspartate receptor-mediated responses in the SN."
https://www.ncbi.nlm.nih.gov/pubmed/8867135
  1. Another paper on how DHA inhibits GABA-receptor mediated responses and enhances glutamatergic synaptic activities (see "EFFECTS OF DHA ON NEUROTRANSMITTERS"); https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3792211/
Could anyone elaborate on this?
Thank you all.
submitted by RelevantBarnacle to benzorecovery

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